Fighting….

[bruschi11]

Can liver disfunction cause brutal change in perception? I feel unreal bad.. I found out amoxicillin + clavulanic acid can cause neuroxicity by damaging liver.. Maybe it dumps b12 or folate?.. I dont know what to do really

Liver dysfunction can cause all sorts of Neuro symptoms. Liver regulates body chemistry like crazy.

 

[bruschi11]

Do you have symptoms of delirium? ALA seems to cause them, it's probably moving iron aluminum and who knows what else, B1 does too. I wanted to ask you which vitamin is most essential for lipid metabolism? B2 ?

I’d say lipid metabolism is based on overall methylation but you can say it’s any one of b2 b6 b12 zinc moly mag folate…. It can be any that is a missing piece in the picture.

Hard to say what my symptoms are but severe severe neurologica stuff it’s really tough. And when I do things wrong I pay for it.

I think Brain needs carbon. Just high quality glutathione production. For an extended period of time. As I work on the liver and use liver health for iron and copper availability. The gluth production really depends on methylation nutrients I list above. You’d think liver flush alone would do it but it helps.

 

[Allavoy]

Can you notify me how I can modify my passphrase?
Maybe I am doing something improperly?
Require your help.
Thank you.

 

[highserotonin90]

I’d say lipid metabolism is based on overall methylation but you can say it’s any one of b2 b6 b12 zinc moly mag folate…. It can be any that is a missing piece in the picture.

Hard to say what my symptoms are but severe severe neurologica stuff it’s really tough. And when I do things wrong I pay for it.

I think Brain needs carbon. Just high quality glutathione production. For an extended period of time. As I work on the liver and use liver health for iron and copper availability. The gluth production really depends on methylation nutrients I list above. You’d think liver flush alone would do it but it helps.

Can you clarify for me if what I understood is correct... molecular hydrogen puts you in a position to consume fats (therefore there is beta oxidation of adipose tissue) but at the same time it puts you in an anti-oxidation condition by sweeping away hydroxyl radicals ROS RNS etc...am I right? Does it put you in alkalosis or acidosis? it should therefore raise cortisol but I don't understand what happens to carbohydrate metabolism. Second question: betaine HCL.. malic acid.. succinic acid.. hydrochloric acid.. can they do the same thing by burning fat? Thank you

 

[bruschi11]

Can you clarify for me if what I understood is correct... molecular hydrogen puts you in a position to consume fats (therefore there is beta oxidation of adipose tissue) but at the same time it puts you in an anti-oxidation condition by sweeping away hydroxyl radicals ROS RNS etc...am I right? Does it put you in alkalosis or acidosis? it should therefore raise cortisol but I don't understand what happens to carbohydrate metabolism. Second question: betaine HCL.. malic acid.. succinic acid.. hydrochloric acid.. can they do the same thing by burning fat? Thank you

I think you’re right and it negates the alkalosis. I don’t know about the acids. I am not a good source for info right now I’m very very sick and my brain is losing a lot that I’ve learned

 

[jimmjimmy]

Hi bruschi

 

[bruschi11]

Hi bruschi

Umm hi?

 

[bruschi11]

I have done 25 htmas. The last 2.25 years worth? All of them have low cobalt. All of them. Very low.

When I first started doing htmas with good health in 2018? Cobalt was top of range or high. Like it was way up there.

When I had antibiotic damage it did start sinking…. Until I got a methylation protocol focusing on FAD and I was liver flushing during that. Cobalt went back up significantly. This was in ‘19-‘20.

It seems like the biggest thing that happens once cobalt goes back up? NRF2 minerals vanadium chromium maybe lithium drop. B5 helped during these times along with cr v li.

Step 1 has to be cobalt. I’ve been reading a lot of Freddd on pheonix rising and and I just feel stupid it took me this long to put it all together.

I think the worst think is dumping iron with low cobalt and that’s been happening. That’s a big reason people need b2 before b1.

B2 zinc/moly for FAD seems crazy important here.

 

[highserotonin90]

I have done 25 htmas. The last 2.25 years worth? All of them have low cobalt. All of them. Very low.

When I first started doing htmas with good health in 2018? Cobalt was top of range or high. Like it was way up there.

When I had antibiotic damage it did start sinking…. Until I got a methylation protocol focusing on FAD and I was liver flushing during that. Cobalt went back up significantly. This was in ‘19-‘20.

It seems like the biggest thing that happens once cobalt goes back up? NRF2 minerals vanadium chromium maybe lithium drop. B5 helped during these times along with cr v li.

Step 1 has to be cobalt. I’ve been reading a lot of Freddd on pheonix rising and and I just feel stupid it took me this long to put it all together.

I think the worst think is dumping iron with low cobalt and that’s been happening. That’s a big reason people need b2 before b1.

B2 zinc/moly for FAD seems crazy important here.

Which HTMA test is more reliable, TEI or ARL? Don't you think that your symptoms are very similar to an encephalomyopathy due to demyelination? If I'm not mistaken, B1 can bind iron (in which organ and tissue I don't know, I hope especially in the liver and brain) Helen said so, while B2 suggested it binds copper. I have never understood whether copper and dietary iron should be avoided during Chelation so as not to create further problems. Bolybdenum competes with sulfur and copper, helps degrade histamine I think and convert sulphite to sulphate.

I believe that the Fred protocol has not been updated since 2018, there are good and interesting ideas in common with Helen... a comparison between the two would have been nice.

 

[bruschi11]

Can you be more clear? Which alkalosis are you talking about, basic or compensatory? I never understood when Helen talked about the CO2 detected via venous/arterial blood gas whether she reflected the CO2 exactly in the cell or whether we should think the opposite. Look at pyruvate dehydrogenase, NAD and B1 are needed if you want to keep it active and make PDK inhibited (DCA, ALA can partially do this). That's why Helen often mentioned that to have an active fat and carbohydrate metabolism you need B1,B3,B5 and maybe even B2. I'm probably missing other things, you can clarify them if you want. Thank you.

Pyruvate dehydrogenase starts with TPP. Which is thiamine based but the problem is thiamine doesn’t just work on its own.

Thiamine needs folate b2 b12 THYROID all to turn on.

This is why the GRJ b2 protocol is so good. You are basically activating b1 with it so you get PDH going. Body makes co2. Everything should be dandy.

Unfortunately one of us here got so bad the last 15 months I think it’s too late for me. I realize whenever I’ve made big progress with nutrients it’s because of PDH comes first.

This sucks. Everything gbold taught was sooo great here. I think the thyroid comes last thing he would say was just THE WORST thing for my head.

I never had any success supplementing selenium so it shied me from thyroid. Little did I know the thing my body needed most was iodine and I was helping myself over the summers getting it in ocean.

 

[bruschi11]

Which HTMA test is more reliable, TEI or ARL? Don't you think that your symptoms are very similar to an encephalomyopathy due to demyelination? If I'm not mistaken, B1 can bind iron (in which organ and tissue I don't know, I hope especially in the liver and brain) Helen said so, while B2 suggested it binds copper. I have never understood whether copper and dietary iron should be avoided during Chelation so as not to create further problems. Bolybdenum competes with sulfur and copper, helps degrade histamine I think and convert sulphite to sulphate.

I believe that the Fred protocol has not been updated since 2018, there are good and interesting ideas in common with Helen... a comparison between the two would have been nice.

TEI or Doctors data. I like oligoscan now too. Oligoscan has been best for my historical issues…. Chromium phosphorus iodine.

 

[balance seeker]

TEI or Doctors data. I like oligoscan now too. Oligoscan has been best for my historical issues…. Chromium phosphorus iodine.

Is that oligoscan truly reliable? How can they evaluate the mineral composition of the body by just merely touching a device on your palm. I am quite skeptical about it.

 

[bruschi11]

Is that oligoscan truly reliable? How can they evaluate the mineral composition of the body by just merely touching a device on your palm. I am quite skeptical about it.

Don’t have much energy to waste arguing for a device. It’s accurate. 4 scans. 2 of them I was stable but struggling. 2 of them horrid dying. The 2 horrid dying sulfur went low and the other historical issues like phosphorus chromium went with it.

 

[bruschi11]

Looks like I’ll be posting here coming up. I’m in rough shape. I’ve never learned more than I have the last few months.

I know that knowing what I know now if I could just go back a year or two I would be able to save my life. I think I might be too late.

I got banned from the ray peat forum for a post that supported their stuff but said “hey it’s not that simple.” I was 100% accurate. These are people with an agenda for what they want to be right while neglecting obvious truths. Very sad and pathetic.

 

[RebelWithACause]

Looks like I’ll be posting here coming up. I’m in rough shape. I’ve never learned more than I have the last few months.

I know that knowing what I know now if I could just go back a year or two I would be able to save my life. I think I might be too late.

I got banned from the ray peat forum for a post that supported their stuff but said “hey it’s not that simple.” I was 100% accurate. These are people with an agenda for what they want to be right while neglecting obvious truths. Very sad and pathetic.

Always been that way there lol. These dudes switched on Ray Peat after he died almost instantly. Which is OK because even Ray Peat said people should think for themselves. Except these motherfuckers still don't think for themselves. They just jumped on the next bandwagon (low vit A). Kinda what gbolduev predicted years ago.

 

[bruschi11]

Always been that way there lol. These dudes switched on Ray Peat after he died almost instantly. Which is OK because even Ray Peat said people should think for themselves. Except these motherfuckers still don't think for themselves. They just jumped on the next bandwagon (low vit A). Kinda what gbolduev predicted years ago.

Glad you’re with me. Not worth talking about them. Just randoms on the internet that don’t truly get it.

I’m getting it all the time and it’s almost silly for me to keep writing “you’re so sick, what you say doesn’t hold any weight.” It’s very frustrating.

The anemia that began with a blood donation to begin 2023 has left me in battle I could never imagine. I’m realizing all the nutrients I gave were just making me worse specifically b12.

The reason is turning on system through b2/ b5 fad and coa just ends up activating a really high b12 in blood causing fast oxidation. TPP turns on hard using up pyruvate and all sorts of nutrients copper calcium. Its definition of fast oxidation in a system that needs electrolyte retention through fad.

Gbold was right it just was so hard to figure everything out that mattered. Nadph/fad balance probably is the key to this. We need both but fad comes first. CoA comes before FAD as beta alanine is needed for glycine reception at the NMda receptor. Also CoA comes first in the Krebs cycle.

So with high b12 you have nadph outweighing fad coa nadh…. All things you need before nadph.

The body runs on enzyme systems. Not individual nutrients. But inflating one single individual nutrient can take down an entire enzyme system if it upregulates the wrong one at the wrong time (nadph).

 

[bruschi11]

Seeing molybdenum really go dead majority of last year in my oligoscans. The nutritionist I’m working with, Meredith Arthur, has been pushing moly on me for a month.

What I didn’t understand is that molybdenum for the SO (sulfite oxidase ) enzyme comes before CDO. As body makes sulfite from just cysteine. I always thought histidine makes sulfite and it does. But it’s not the only source.

I feel so stupid.

 

[RebelWithACause]

Glad you’re with me. Not worth talking about them. Just randoms on the internet that don’t truly get it.

I’m getting it all the time and it’s almost silly for me to keep writing “you’re so sick, what you say doesn’t hold any weight.” It’s very frustrating.

The anemia that began with a blood donation to begin 2023 has left me in battle I could never imagine. I’m realizing all the nutrients I gave were just making me worse specifically b12.

The reason is turning on system through b2/ b5 fad and coa just ends up activating a really high b12 in blood causing fast oxidation. TPP turns on hard using up pyruvate and all sorts of nutrients copper calcium. Its definition of fast oxidation in a system that needs electrolyte retention through fad.

Gbold was right it just was so hard to figure everything out that mattered. Nadph/fad balance probably is the key to this. We need both but fad comes first. CoA comes before FAD as beta alanine is needed for glycine reception at the NMda receptor. Also CoA comes first in the Krebs cycle.

So with high b12 you have nadph outweighing fad coa nadh…. All things you need before nadph.

The body runs on enzyme systems. Not individual nutrients. But inflating one single individual nutrient can take down an entire enzyme system if it upregulates the wrong one at the wrong time (nadph).

I don't get everything either but I do know that all these diets are a means to an end. A way to make your specific health better. Doesn't mean this works for everybody. And that part is often forgotten and/or ignored. It's very hard to not fall into that type of thinking. I used to think the same. I saw some stuff specifically worked for me. But now I am better I cannot do those same things anymore. And some people are probably already at the point I am now. If I would recommened them what I did back then to them they would also feel bad and it wouldn't do anything for them.

 

[bruschi11]

Seeing molybdenum really go dead majority of last year in my oligoscans. The nutritionist I’m working with, Meredith Arthur, has been pushing moly on me for a month.

What I didn’t understand is that molybdenum for the SO (sulfite oxidase ) enzyme comes before CDO. As body makes sulfite from just cysteine. I always thought histidine makes sulfite and it does. But it’s not the only source.

I feel so stupid.

Been taking 75-150mcg of moly more days than not. I can’t tell you exacts but I have been taking it. Thing is it has gone nowhere in my oligoscans. Low end of range in hair.

There were times in past where giving moly didn’t do anything then I’d go high with it 3-500mcg short term and things would change.

Bmaa toxicity is what I’ve had for 4 years. I’ve been dying from it for about a year. Bmaa in my opinion competes with beta alanine and alanine.

Alanine is produced in the cysteine pathways according to AI. Found that out yesterday. Where moly is needed everywhere so I’m sure next to b5 active b6 (zn/mg/b2), moly plays big role in production of alanine.

I was wrong for a long time and it may have cost me my life. I thought moly comes after histidine in CDO pathway and it does. BUT in CBS pathway active b6 is reliant on moly prior to histidine raising. Active b6 without histidine still produces sulfite which moly is needed for.

Heck nrf2 produces cysteine from cystine so that pressures moly as well.

This really sucks as I’ve been dying for 18 months severely for a year worse than ever for about 6 months now.

And the biggest piece likely was a mineral that I have had profound experiences. Several times between late 2019 and mid 2022.

 

[bruschi11]

S-sulfocysteine is the problem that occurs and causes ALT liver enzyme to elevate. But it’s a sign of something bigger.

S-sulfocysteine is only gonna accumulate if sulfite accumulates or there is no nadph.

Nadph went down when inositol went down two years ago from Phos choline IVs. Nadph lowers s sulfocysteine (SSC) converting it back to cysteine. BUT without moly and elevated sulfite, sulfite will just bring cysteine back to SSC. Pressuring nadph more.

Now does SSC upregulate nadph? Nadph clears SSC. Nadph was not upregulated until I started pushing b2 and b5. These use b12 to raise nadph. But is nadph just extra high because the body is trying to clear SSC? Or is it just the high b12 making nadph extra strong.

We know nadph is high because of threonine elevation in aminos.

We are going to stop pushing other nutrients just load moly. Take chromium with it (moly antagonizes it). Much less b2 inositol choline b5 .

I have to realize moly saved my life in both 2021 and 2022. Unfortunately I never followed enough to say “I have to keep moly sufficient indefinitely”. And it bit me bad