- Deleted by zancek0
- Reason: have to change it
ANYWAYYYYY,
I've been running a group for people experiencing long-term "low E2" symptoms (of course, this is based mostly on vibes) after various kinds of interventions directly affecting aromatase or ERa/ERb ratio (let's assume talking about such a ratio even makes "theoretical/conceptual" sense). Let's say we call this group "PAIS" (post aromatase inhibitor syndrome) since most of these people are those that got hurt by aromatase inhibitors (but there's also people with long-term low E2 symptoms after tamoxifen, masteron, improperly handled TRT etc.). I've thus far met and talked to about 20 people (from different forums) that fit this description. My conclusion is that there's as much likeness (in terms of blood work, symptoms, background etc.) between cases as there is among PFS (and PSSD, PAS, lion's mane, ashwaganda, saw palmetto-syndrome etc.) cases. Meaning - it's not technically correct to say that it is a disease of its own (a totally unique imbalance). If we studied these syndromes long enough, we could find some underlying patterns (on hs Gbold and others often talked about "types" of PFS, PSSD etc.; Gbold even created protocols for 2 specific types). I did observe some patterns but, of course, nothing conclusive and, as I said, too much similarities to other syndromes.
I've been running a group for people experiencing long-term "low E2" symptoms (of course, this is based mostly on vibes) after various kinds of interventions directly affecting aromatase or ERa/ERb ratio (let's assume talking about such a ratio even makes "theoretical/conceptual" sense). Let's say we call this group "PAIS" (post aromatase inhibitor syndrome) since most of these people are those that got hurt by aromatase inhibitors (but there's also people with long-term low E2 symptoms after tamoxifen, masteron, improperly handled TRT etc.). I've thus far met and talked to about 20 people (from different forums) that fit this description. My conclusion is that there's as much likeness (in terms of blood work, symptoms, background etc.) between cases as there is among PFS (and PSSD, PAS, lion's mane, ashwaganda, saw palmetto-syndrome etc.) cases. Meaning - it's not technically correct to say that it is a disease of its own (a totally unique imbalance). If we studied these syndromes long enough, we could find some underlying patterns (on hs Gbold and others often talked about "types" of PFS, PSSD etc.; Gbold even created protocols for 2 specific types). I did observe some patterns but, of course, nothing conclusive and, as I said, too much similarities to other syndromes.