Another Theory for PFS ( Andregenic receptor )

[Namelk]

I have already seen 2 cases of accutane that have been cured with metronidazole and paromomycin, the one mentioned so and another in an accutane forum, but I do not find these antibiotics in my city and need revenue, it would be nice to know other things with similar effects, healed after the end of treatment

 

[jinstewart]

Did ANYONE try this in the end? Don't let this one die... :/

EDIT - we need an official Hackstasis experiments crew I swear...

If my SJW run doesn't fix me on snapback I'll do this. Ordering the Silodyx now.

Probably end of September is the earliest I'll start. It'll be 400mg R-Andro and 4md Silodyx every morning and I'll try it for 3-4 weeks I guess.

 

[zancek0]

antagonists alfa 1 receptor, agonism of alfa 2 receptor=outcome will be raised prolactine, gh, low acth,insulin,cortisol, so very low libido and feeling seedation, better night erections and erections from touching and seeing-drugs which work on this is for example tizanidine, and maybe as D4n mention minoxidil

agonism alfa 1 receptor and antagonism alfa 2- have opposite way of work, they increase libido (yohimbine is alfa 2 antagonists), sweating,dilated pupils

cocaine, amphetamine are alfa 1 agonists

I know this is old but I am thinking about it: why would less noradrenaline lead to raised prolactin and so on (agonism of alpha 2 receptor would inhibit release of noradrenaline, leading to more dopamine?)...
Is it a matter of tonic/phasic balance? When dopamine to noradrenaline conversion runs smoothly, the body has constantly properly sensitive dopamine receptors?
Or is it something like: less noradrenaline = less adrenaline = more sensitive beta receptors = increased requirement for calcium?

 

[ruprmurdoch]

I know this is old but I am thinking about it: why would less noradrenaline lead to raised prolactin and so on (agonism of alpha 2 receptor would inhibit release of noradrenaline, leading to more dopamine?)...
Is it a matter of tonic/phasic balance? When dopamine to noradrenaline conversion runs smoothly, the body has constantly properly sensitive dopamine receptors?
Or is it something like: less noradrenaline = less adrenaline = more sensitive beta receptors = increased requirement for calcium?

that info is result of my research in web

 

[RebelWithACause]

DHT seems the main protector for males against stress. I notice when my cortisol goes up so does my DHT. Maybe also add estrogen to it. It makes you feel good during stress pretty much or at least lower the feelings of stress. Same for 5ar hormones like allo which I think is more focussed on the brain to keep mental stress at bay.

DHT more focussed on sexual function DURING stress. This is why on finasteride when I was stressed I could not get a boner. But in a low stress environment I had no problems. With normal DHT I can always get a boner. Even during extreme stress I can get a boner when DHT levels are good. DHT/5ar hormones always keep me sexually potent so to speak doesn't matter what happens.

So when you take finasteride you just have uncontrollable stress physical and mental. Nothing opposes it and you lose your sexual function. Or when you get PFS which pretty much is still Finasteride in your body lowering 5ar because it stops its own metabolism. Or it epigenetically did so.

Remember the guy who said he was pacing up and down the road because he felt so stressed. Extreme stress. I also paced a lot in that time just couldn't sit still from feeling stressed. Just complete uncontrollable stress. And I was also not eating good in that time because of that stress and this made things even worse. In this case I think fasting is not good for PFS except maybe to lower cortisol level over time? But I think you just end up more stressed after.

So the idea DHT is bad or something is just stupid... but yea most people know that here already.

Of course there is also people with the opposite where they have no anxiety/stress and too much DHT/androgen effects. Helen explained those two cases too