Aleksandr
Well-Known Member
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As we kind of know, different minerals have different effects on oxidation. The more stimulatory, the faster your oxidation and the more sedative, the slower your oxidation. It seems phosphorous is the most stimulatory, and calcium the most sedative. The 'transitional minerals / vitamins' appear to be balancing - if you're too slow, they speed you up and vice versa.
Stimulatory Minerals / Vitamins:
Phosphorus
Sodium
Potassium
Iron
Manganese
Selenium
A
E
B1
B6
B10
Sedative Minerals / Vitamins: :|
Calcium
Magnesium
Zinc
Copper
Chromium
D
B2
B12
Choline
Transitional Minerals / Vitamins:
Zinc
B5
On Food:
The mineral content of foods will vary according to that of the soils in which the food is grown, as well as processing methods and type of cooking utensils used in prepering it (copper, aluminium, etc.)
Protein has the highest Specific Dynamic Action (SDA), and therefore produces the greatest increase in the metabolic rate (sympathomimetic). Part of the effect is due to the calcium and magnesium excretion produced by protein. High density proteins have a higher SDA than low density proteins, with beef having agreater action than fish or fowl, and vegetable protein having the lowest SDA.
Hard water, which has a high total hardness is usually alkaline. The sedative minerals calcium and magnesium are also usually high relative to the stimulatory minerals, and therefore, is considered sedative (parasympathetic).
Softened water is considered stimulatory (sympathetic) as it has low total solids and is generally acidic while dominant in the stimulatory minerals, especially sodium.
On Drugs:
Drugs can be categorized by their sympathomimetic or parasympathomimetic action, which mimics sympathetic or parasympathetic nervous system activity.
Some of the sympathetic inducing drugs include; epinephrine, phenylephrine and methoxamine. Other drugs produce a sympathetic action by affecting neurotransmitter release. These include ephedrine, tyramine and amphetamines.
Sympatholytic drugs can be considered sedative in that they block sympathetic activity centrally or peripherall by inhibiting or blocking neurotransmission. Centraly acting sympathetic inhibitors include clonidie and methyldopa. Reserpine and rauwolfia are alkaloids that prevent the synthesis and storage or norepinephrine, while guanethidine blocks its release. Alpha and beta receptor blockers are commonly used in the treatment of hypertension.
Parasympathomimetic drugs include acetycholine, muscarine, pilocarpine, methacholine and carbamylcholine. These are commony used in the treatment of neurological or neuromuscular disturbances such as myasthenia gravis.
Classification of Disease Processes:
In order to be able to use this information, we should become aware of disease conditions that manifest as sympathetic or parasympathetic disorders. The following is a partial list of conditions that can be classified accordingly. This list is not complete and there are always exceptions.
Sympathetic Diseases:
Anxiety
Arthritis (rheumatoid)
Allergies (histamine)
A. L. S.
Hypertension
Hyperthyroid
Hyperadrenia
Hodgkins
Leukemia
Infections (bacterial)
Myasthenia Gravis
Multiple Sclerosis
Ulcers (peptide or duodenal)
Diabetes (juvenile)
Parasympathetic Diseases:
Arthritis (osteo)
Allergines (low histamine)
Asthma
A. I. D. S.
Anorexia
Fungus
Hypotension
Hypothyroid
Hypoadrenia
Infections (Viral)
Lupus
P. M. S.
Yeast
Ulcers (Gastric)
Diabetes (adult onset)
Nutritionally induced deficiencies:
Often brought about by megadosing. The response can be interference with the utilization of another nutrient, thus becoming an antivitamin or antimineral. If continued for long periods this can induce a deficiency of another nutrient. I.e. if a patient is experiencing calcium deficiency symptoms and is not responding to 800 or 1000 mg of calcium supplementation per day, the clinicians first inclination is to increase the dosage, perhaps two or three times this amount. This may improve the patients symptoms but, even after several months, reduction in calcium intake will result in an almost immediate return of symptoms. To maintain the patient in an asymptomatic state, the dosage requirements will usually increase with time rather than decrease. If the synergists and antagonists of calcium are considered, such as the addition of vitamin D, magnesium, or copper, and the reduction of vitamin E, vitamin A, potassium, phytic acid nd oxalic acid foods, the patient may respond to only 400mg of calcium supplementation per day.
This summary was taken from an article at the trace elements website:
http://www.traceelements.com/Docs/Nutrient%20Interrelationships-Minerals-Vitamins-Endocrines.pdf
They have other articles too which are helpful in interpreting your own hair test results.
As we kind of know, different minerals have different effects on oxidation. The more stimulatory, the faster your oxidation and the more sedative, the slower your oxidation. It seems phosphorous is the most stimulatory, and calcium the most sedative. The 'transitional minerals / vitamins' appear to be balancing - if you're too slow, they speed you up and vice versa.
Stimulatory Minerals / Vitamins:
Phosphorus
Sodium
Potassium
Iron
Manganese
Selenium
A
E
B1
B6
B10
Sedative Minerals / Vitamins: :|
Calcium
Magnesium
Zinc
Copper
Chromium
D
B2
B12
Choline
Transitional Minerals / Vitamins:
Zinc
B5
On Food:
The mineral content of foods will vary according to that of the soils in which the food is grown, as well as processing methods and type of cooking utensils used in prepering it (copper, aluminium, etc.)
Protein has the highest Specific Dynamic Action (SDA), and therefore produces the greatest increase in the metabolic rate (sympathomimetic). Part of the effect is due to the calcium and magnesium excretion produced by protein. High density proteins have a higher SDA than low density proteins, with beef having agreater action than fish or fowl, and vegetable protein having the lowest SDA.
Hard water, which has a high total hardness is usually alkaline. The sedative minerals calcium and magnesium are also usually high relative to the stimulatory minerals, and therefore, is considered sedative (parasympathetic).
Softened water is considered stimulatory (sympathetic) as it has low total solids and is generally acidic while dominant in the stimulatory minerals, especially sodium.
On Drugs:
Drugs can be categorized by their sympathomimetic or parasympathomimetic action, which mimics sympathetic or parasympathetic nervous system activity.
Some of the sympathetic inducing drugs include; epinephrine, phenylephrine and methoxamine. Other drugs produce a sympathetic action by affecting neurotransmitter release. These include ephedrine, tyramine and amphetamines.
Sympatholytic drugs can be considered sedative in that they block sympathetic activity centrally or peripherall by inhibiting or blocking neurotransmission. Centraly acting sympathetic inhibitors include clonidie and methyldopa. Reserpine and rauwolfia are alkaloids that prevent the synthesis and storage or norepinephrine, while guanethidine blocks its release. Alpha and beta receptor blockers are commonly used in the treatment of hypertension.
Parasympathomimetic drugs include acetycholine, muscarine, pilocarpine, methacholine and carbamylcholine. These are commony used in the treatment of neurological or neuromuscular disturbances such as myasthenia gravis.
Classification of Disease Processes:
In order to be able to use this information, we should become aware of disease conditions that manifest as sympathetic or parasympathetic disorders. The following is a partial list of conditions that can be classified accordingly. This list is not complete and there are always exceptions.
Sympathetic Diseases:
Anxiety
Arthritis (rheumatoid)
Allergies (histamine)
A. L. S.
Hypertension
Hyperthyroid
Hyperadrenia
Hodgkins
Leukemia
Infections (bacterial)
Myasthenia Gravis
Multiple Sclerosis
Ulcers (peptide or duodenal)
Diabetes (juvenile)
Parasympathetic Diseases:
Arthritis (osteo)
Allergines (low histamine)
Asthma
A. I. D. S.
Anorexia
Fungus
Hypotension
Hypothyroid
Hypoadrenia
Infections (Viral)
Lupus
P. M. S.
Yeast
Ulcers (Gastric)
Diabetes (adult onset)
Nutritionally induced deficiencies:
Often brought about by megadosing. The response can be interference with the utilization of another nutrient, thus becoming an antivitamin or antimineral. If continued for long periods this can induce a deficiency of another nutrient. I.e. if a patient is experiencing calcium deficiency symptoms and is not responding to 800 or 1000 mg of calcium supplementation per day, the clinicians first inclination is to increase the dosage, perhaps two or three times this amount. This may improve the patients symptoms but, even after several months, reduction in calcium intake will result in an almost immediate return of symptoms. To maintain the patient in an asymptomatic state, the dosage requirements will usually increase with time rather than decrease. If the synergists and antagonists of calcium are considered, such as the addition of vitamin D, magnesium, or copper, and the reduction of vitamin E, vitamin A, potassium, phytic acid nd oxalic acid foods, the patient may respond to only 400mg of calcium supplementation per day.
This summary was taken from an article at the trace elements website:
http://www.traceelements.com/Docs/Nutrient%20Interrelationships-Minerals-Vitamins-Endocrines.pdf
They have other articles too which are helpful in interpreting your own hair test results.
