PFS almost cured

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hairsuit

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Helen said:
you need to understand that fin inhibits DHT at very very small doses.

that is why I said if someone keep taking microdoses , that someone will have constant tiny binding to NADPH<

and this works as a stilt for PFS.


like 0.02 pill of FIN , would probably inhibit DHT as much as 5mg pill


So it is very very hard to find a dosage which will inhibit DHT just right.

this is the problem with FIN in the first place.

if people took it in tiny amounts in the first place, they probably would not have problems at all while on it


as I said, I had high DHT, very high , and my sex drive was meh,

but when I went on FIN, my sex drive my muscles, EVERYTHING Was better, Times over. since fin lowered DHT

and this increased test and estro action
Click to expand...
Sooooooooo, fin crumbs for everyone!!!!!!
 
Helen

Helen

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hairsuit said:
Sooooooooo, fin crumbs for everyone!!!!!!
Click to expand...





NO may be diluted in water. but still this is for high DHT cases


when people have hairloss, they can use fin, but they need to get DHT just slightly lower,

to do that you need to dilute fin or duta and take tiny amounts and measure blood to see that your DHT is fine.

this way it works like that.
 
Helen

Helen

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@hairsuit I found another dude. who did the same as this guy

I was in a similar state as you describe for a full year after stopping. I noticed that if I took a small dose then stopped for a week or two I would get Moring wood again for a period until a "crash" when it went back to like before. I kept doing that and enjoying my week or 2 of erections at a time and eventually didn't have to keep taking it anymore and was back close to normal. Maybe I just didn't taper off long enough originally.



May be the problem was that people on propecia forum, took it and weaned off actually by taking finasteride and lowering it which does not work since finasteride will inhibit DHT even at low dosages. same as higher dosages

but may be proper weaning off is done by taking it and stopping for weeks. this way you are slowly weaning off of it.

and the body has time to get rid of overexpressed ARs.
 
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Helen

Helen

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Like if you take something in big amounts, and then stop quickly, DHT goes up right away in big amount. and this will not give time for the body to downregulate number of ARs.

and now body has to shut down ARs with methylation, and boom testosterone does not work.

but if you take little FIN , very little, then this changes of DHT spike are much smaller, and longer.

and this way body can adjust the ARs and downregulate the actual NUmber of ARs and keep methylation put.

I think this has merit , may be.

And this is probably the problem of proper weaning off the finasteride.
 
TubZy

TubZy

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Helen said:
@tanedout Also if ARs were downregulated as you said in the 2 posts above, he would not feel like total crap everysingle time his DHT goes up

but he feels better when he lowers DHT. and bad when it goes back up.

So he failed to find small enough dosage of fin to keep DHT a little down all the time.

instead he takes fin every 2 weeks, when DHT creeps up
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I guess the thing is that many people who quit finatseride also don't crash meaning they quit fin and a few weeks later return to the state they were at prefin like high libido again, oily skin, hair loss etc. while others like us crash hard, we still don't know why that happens like why some ppl get PFS and others don't
 
Helen

Helen

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Boris said:
I guess the thing is that many people who quit finatseride also don't crash meaning they quit fin and a few weeks later return to the state they were at prefin like high libido again, oily skin, hair loss etc. while others like us crash hard, we still don't know why that happens like why some ppl get PFS and others don't
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Dude, I think this happens, since some people can break down DHT after it goes back up.

and PFS people cant break it down, when it goes back up.

this is why all PFS people have low 3 adiol g. meaning that glucorinadation , does not work.


So if normal person takes FIN , and then gets off.

then DHT goes up slowly in normal person, since as it goes up the breaking down of DHT also increases

and in PFS person, as DHT goes up, breaking down does not happen, and body has to use methylation to fucking Shut every thing down since DHT rise is TOO FAST and receptors are overexpressed also.

So the problem with PFS is in the liver and overexpressed ARs

I think I understand how one dose of fin can help it. taking every 2 weeks. , but that dose has to be very very small. and taken for a year may be
 
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Helen

Helen

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you need to downregulate numbers of ARs , if you take tiny dose of fin, and then quit for 2 weeks, for the body it is like taking DHT. since it is the increase in DHT which will downregulate AR expression.


If people take HUGE dose of fin, DHT goes to zero. body adjusts numbers of receptors, like the cell will have instead of 10 receptor proteins , it will have 30.

then you quit fin and DHT goes up very fast. but the cell has 30 receptors now. and body cant manage fast enough to downregulate number of receptors

So it just shuts down ARs with methylation, and ARs where testosterone is DOnt work.



so now you are stuck with the same 30 overexpressed receptors and increased methylation to stop the ARs

so if you lower DHT. this will stop methylation, and then if you increase DHT gradually. slowly , constantly, this will give time for the body to downregulate NUMBER of ARS. and keep methylation put.


I guess the same thing can be done, by just killing methylation for a bit.( i was thinking about it, lets say with NIACIN)) or feeding high doses of DHT which wil overwhelm the methylation ( Cdnuts protoclol, r andro cycles)

and since methylation is not there. boom, numbers of ARs will have to downregulate. and then you quit niacin , high dose

and you are back to normal.
 
W

wuf

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Helen said:
Dude, I think this happens, since some people can break down DHT after it goes back up.

and PFS people cant break it down, when it goes back up.

this is why all PFS people have low 3 adiol g. meaning that glucorinadation , does not work.


So if normal person takes FIN , and then gets off.

then DHT goes up slowly in normal person, since as it goes up the breaking down of DHT also increases

and in PFS person, as DHT goes up, breaking down does not happen, and body has to use methylation to fucking Shut every thing down since DHT rise is TOO FAST and receptors are overexpressed also.

So the problem with PFS is in the liver and overexpressed ARs

I think I understand how one dose of fin can help it. taking every 2 weeks. , but that dose has to be very very small. and taken for a year may be
Click to expand...
So, who is going to be the guinea pig?!
Come on, out balls who has them!...
 
W

wuf

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880
Helen said:
you need to downregulate numbers of ARs , if you take tiny dose of fin, and then quit for 2 weeks, for the body it is like taking DHT. since it is the increase in DHT which will downregulate AR expression.


If people take HUGE dose of fin, DHT goes to zero. body adjusts numbers of receptors, like the cell will have instead of 10 receptor proteins , it will have 30.

then you quit fin and DHT goes up very fast. but the cell has 30 receptors now. and body cant manage fast enough to downregulate number of receptors

So it just shuts down ARs with methylation, and ARs where testosterone is DOnt work.



so now you are stuck with the same 30 overexpressed receptors and increased methylation to stop the ARs

so if you lower DHT. this will stop methylation, and then if you increase DHT gradually. slowly , constantly, this will give time for the body to downregulate NUMBER of ARS. and keep methylation put.


I guess the same thing can be done, by just killing methylation for a bit.( i was thinking about it, lets say with NIACIN)) or feeding high doses of DHT which wil overwhelm the methylation ( Cdnuts protoclol, r andro cycles)

and since methylation is not there. boom, numbers of ARs will have to downregulate. and then you quit niacin , high dose

and you are back to normal.
Click to expand...
Following your logic:
you sat, give DHT to kill methilation. --> high doses of DHT which wil overwhelm the methylation ( Cdnuts protoclol, r andro cycles)
and since methylation is not there. boom, numbers of ARs will have to downregulate...WHY? DHT is there at big quantity, why should ARs downregulate since a lot of DHT is there?
 
Helen

Helen

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wuf said:
Following your logic:
you sat, give DHT to kill methilation. --> high doses of DHT which wil overwhelm the methylation ( Cdnuts protoclol, r andro cycles)
and since methylation is not there. boom, numbers of ARs will have to downregulate...WHY? DHT is there at big quantity, why should ARs downregulate since a lot of DHT is there?
Click to expand...


first of all, people have DHT in different quantity. from low to high.


Second of all, ARs are silenced by methylation. where DHT is.

so if you take some DHT, numbers of ARs will decrease.

If you lower DHT , methylation will stop. and then if you slowly increase DHT , then this will downregulate number of ARs
 
Helen

Helen

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I wonder if people with high DHT feel better on NIACIN.

like if pound on niacin in grams this should stop methylation, and as methylation would be stopped.

then the body will have to downregulate the number of ARs since it basically has no protection anymore from high DHT levels.



as as number of ARs downregulate , then less methylation would be needed after
 
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RebelWithACause

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Guy still has watery semen, weak orgasm after 1 year of doing that. Look at last post he posted. I think he did fix mental sides though - at least he does not write about that.
 
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