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Reta just Turns on fat oxidation too hard along with the GLP1 sides. I’m on the tirzepatide now and I just think it uses up glucagon too much when that is already the issue low glucagon. Like these drugs are for normal people. Not guys with severe cfs for years. They’re trying to use...

Bile over last 3 days since starting carnitine keeps getting more and more perfect. Very bad neurologically. I think I took too much iodine the other day. But the tirzepatide I think is not right for what I’m doing. I think it’s acting like b1 in a system that badly needs meb12 . Lithium is...

Basically cps1 needs glucagon for inositol. Which starts with carnitine. But needs ERb (nad e2 and others) to use carnitine to make glucagon . Iodine seems to come post carn

i really don’t know if tirz whatever it’s called is right for me but it might. Insulin has to drop. When insulin drops, chromium is retained in bone. Same with glycogen rising I THINK. Pretty sure chromium goes to liver and is pissed out in low glycogen high insulin conditions. I may not be...

This is the pathway to life I see it as…. Attached is the image. We gotta get down these pathways to make BA. It all really starts with co2 and ATP. But cps1 which is sirt5 regulated opens these up down to orotate. But fmn is needed there too to bring orotate westward. Fmn we know is b2...

the choline cycle is what we want for glycine. But we also gotta think inositol for co2 for beta alanine. It’s really Ray peat Gbold. Co2+ atp for beta alanine + nad/choline for glycine = nmda

Reta is gonna be known as a savior for many. A killer for others. I think it blows out choline inositol as it dumps copper and iron. And no co2 no glycine for copper using so no nmda receptor. It’s a bad drug for those with chronic illness and electrolyte issues. I might need a week or two...

I think it goes in order… Meb12 Adob12 Carnitine (not sure which one yet maybe not fumarate maybe acetyl) Choline Methylfolate Lithium Iodine As those the most direct ways to pushing Krebs cycle. I really have high hopes for ss31 and mots c. But I honestly don’t think I’m gonna feel...

Reta is a lot. Too much fat oxidation really destroying the brain. I get it tho. Fat oxidation huge part of Freddds protocol. I have libido now and I had raging libido on carnitine over winter for a few days. It’s clear fat oxidation is taking down fluoride. In order…. Meb12 + atp= Ado b12...

Fatty liver. 4 years of elevated liver enzymes. I’m 70 lbs overweight. It all happened pretty quick in 2020 the fat gain. I stayed in the 215 to 230 lb range which is bad but not terrible for a few years. But the last 2 years i ballooned to 255 waking up this morning. The fatty liver is biggest...

Mots-c woke me up a lot today after 48 hours off and boron rising again. It is the key to a lot it seems. I think it just feeds pyruvate giving PDH the fuel it needs. Inositol raises somehow. Co2 happens. Choline and meb12 need to happen. Choline in heavier quantities than meb12. I’m starting...

i hope it’s not too late. Choline is a huge a monster really. It’s the master lipid. Cuz yea I’m making glycine with it. I feel it. Glycine is that feeling. But I’m really bad. We need b6 active next to the glycine. B6 activity will allow serine to raise . The plan now has really shifted...

Basically I’m saying I can’t raise chromium with cr supplements. I have to fix chromium through metabolism and diet somehow . Ultimately the hormone are huge for it as estrone sulfate retains chromium in body. Sulfation is huge here. I think I’m having a hard time sulfating my estrone.

We wanna raise fad with chromium. We wanna raise fmn with inositol/iodine. Inositol iodine without chromium is fmn without fad so it’s basically b6 no folate. no glycine serine. Chromium is key to folate as iron as fe2 is the key to keep folate lifted as Histidine recycles folate (Gbold zinc...

I think chromium is the liver/ pancreas issue. And inositol depletion did cause this. But fixing inositol with mots-c isn’t fixing it. Although I do believe the mots-c is a needed ingredient to fix this. You wonder if PC IVs did cause the initial inositol depletion in mid 2022. I basically...

I have been drinking coffee almost daily. And the iodine drops do go with that. Potassium just dead the worst I’ve ever seen it.

Potassium issues very bad very scared just bad bad bad bad Mots C needs to happen. B12 does too. I think. Testo. I But from there it’s like iodine lithium electrolytes. And I can’t even get past iodine to lithium. Iodine is dropping hard from what I’m doing. The most-c really must be...

Beta alanine (motsc-> inositol) For nmda needs Glycine (choline ) Taurine (nadph… iodine) Iodine finally went in yesterday and with that sulfur finally went up and I think that last step was gettting to taurine.

Body runs on sugar and electrolytes. Beta alanine / inositol/ b5 / thyroid—- sugar Choline/ glycine/ chloride/ mg/ k - electrolytes

Yup…. Back to nmda. Inositol = beta alanine Choline = glycine Beta alanine receives glycine at nmda. But so does taurine which needs nadph… many ways to nadph. Is iodine required?