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Experimental Hair Loss Options
- Thread starter TubZy
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What type of loss have you? I was thinking about trying that S5 cream, but i'm a diffuser, probably hard to apply, isnt it?
MNK99
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As a teen, I had a slight line in the front of my hair, never noticed it till I became vainer years later in my 20s. Noticed hair all over my hotel bed, a few nights after trialling Adderall.
Diffuse hair loss --telogen effluvium. No real recession then (nor now, maybe slightly but just volume loss) Still crazy thick hair -- at that point. I was traumatized tho! Finally found out wtf was wrong with me and*** the med didn't screw my mind up, but it did hurt my hair.
Quit meds. Changed to a smoother one, and used s5 once a day and sometimes twice but noticed slightly less hard and also less hyposexual.
I'd wake up or stay awake, shower, shave, apply s5 in my diffuse "parts", and styled it with that, blowdried it, and applied some 'fixing hairspray/texture cream'. Its for minor loss really.
-I think AMPS set off epigenetic changes -- turning off hair loss genes. Obv can't prove this.
-All was cool until I tried another psyche med -- I mtnd hair mostly, until I tried anticonvulsant. Hence, fin. Hence, current issues. Summary: S5 works when diffuse hair still volumous. Pairs well with toppik, possibly minox but I was too scared then to use it > 1 day.
SO in short 90% of my hairloss was Adderall and Lamotrigine. Dexedrine is cool with me. But because of previous and then newer hairloss - I foolishly tried finasteride. Big fuckking mistake.
Diffuse hair loss --telogen effluvium. No real recession then (nor now, maybe slightly but just volume loss) Still crazy thick hair -- at that point. I was traumatized tho! Finally found out wtf was wrong with me and*** the med didn't screw my mind up, but it did hurt my hair.
Quit meds. Changed to a smoother one, and used s5 once a day and sometimes twice but noticed slightly less hard and also less hyposexual.
I'd wake up or stay awake, shower, shave, apply s5 in my diffuse "parts", and styled it with that, blowdried it, and applied some 'fixing hairspray/texture cream'. Its for minor loss really.
-I think AMPS set off epigenetic changes -- turning off hair loss genes. Obv can't prove this.
-All was cool until I tried another psyche med -- I mtnd hair mostly, until I tried anticonvulsant. Hence, fin. Hence, current issues. Summary: S5 works when diffuse hair still volumous. Pairs well with toppik, possibly minox but I was too scared then to use it > 1 day.
SO in short 90% of my hairloss was Adderall and Lamotrigine. Dexedrine is cool with me. But because of previous and then newer hairloss - I foolishly tried finasteride. Big fuckking mistake.
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Wow. thanks for finding this study. Now we have to figure out why this histidine thing is low. I found studies where people took copper zinc cysteine histidine and had amazing hair growth. Also there is a pattent of using copper histidine glycine.
So there must something else causing PE,ED. Otherwise all balding ones MBP and TE would have ED.
Damn it, there is so much information and so much more to discover while other people still believe in a mysterious balding gene and take finashitsteride prescribed by a dump doc
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well by lowering crh, i guess you will stop histamine release and spare histidine. but the thing is that crh is released where there is need for cortisol, and cortisol is made sensitive by zinc finger. So any part of zinc finger missing and you will have high crh release. This is why the balance between cysteine histidine and zinc so important.
Some fast oxidizers are helped by increasing methionine , copper, magnesium potassium
and slow oxidizer I guess by increasing manganese plus zinc plus and histidine
Some fast oxidizers are helped by increasing methionine , copper, magnesium potassium
and slow oxidizer I guess by increasing manganese plus zinc plus and histidine
Thanks, that makes sense.
Do you know what could mean lowered alcohol tolerance? I have always had a "weak head", bu last time I was drinking with my firend after one single beer I felt quite drunk and tired. When I started drinking the second one I coulnd't really finish that, I just felt like my body does not want that. After 1-2hours when I came back home I got a headache that lastet maybe 1,5h. That was very weird as e.g 2 years ago(I haven't TE had yet) I could dring 4 beers, some vodka and other drinks and I didn't 'die'. That smells sth wrong is with that hista- stuff. I am going to a doctor this week to ask him about that, but I am quite sceptical as for now everything I got from them was: "finasteride", "that's cus of genes", "...but u are completely healty", etc.
Do you have any idea what could induce such a reaction?
Canari
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What a surprise!!! Do you mean ALL slow oxidizers need to chelate copper and need to take zinc and manganese? Can't slows have either low or high copper? Or do you distinguish between 1-2-3-4s?
https://www.ncbi.nlm.nih.gov/pubmed/12970166
CRH induces secretion of TSH. That would mean increased CRH causes hypertyroid like symptoms. But this does not makes much sense with slow oxidizers
CRH induces secretion of TSH. That would mean increased CRH causes hypertyroid like symptoms. But this does not makes much sense with slow oxidizers
tallglass13
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Cortisol insensitivity makes so much more sense, than the testosterone theory of Male pattern balding. If you look at bald males, it is obvious that some form of stress has caused them to bald. Whether it be excessive masturbation or sex, alcohol and weed, or excessive exercise. I like to look at UFC fighters. Some of them started out with a full head of hair. but as the years go by, and they continue to fight and train hard, some begin to get male pattern balding. Despite these guys being is fantastic shape, they still lose their hair. I listened to the Ray Peat clip on youtube when he was asked about the cause of male pattern balding. Near the end of the clip, he states that Cortisol is up, and with the low metabolic rate, cholesterol goes more towards Cortisol, than what is optimal to go towards DHEA, testosterone, and progesterone, things that grow the hair. Any thoughts on that @gbolduev
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JonnyCraig
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gbolduev,
Top EAA deficiencies in patients with androgenic apolecia:
1. Histidine 97.08%
2. Leucine 97.06%
3. Isoleucine 73.53%
4. Lysine 29.41%
5. Methionine 20.59%
6. Phenylalanine 20.59%
Top non essential AA deficiencies in patients with androgenic alopecia:
1. Alanine 91.18%
2. Cysteine 55.58%
3. Glutamine 26.47%
4. Proline 23.53%
5. Glycine 14.71%
Very interesting how Isoleucine and Leucine is so high there.
Should we try supplementing with L-Histidine to raise histidine levels? Also Alanine, Cysteine, Lysine, Glutamine, etc?
From this study, it sounds like BCAA's are definitely beneficial for hair, so BCAA's to get the Leucine and Isoleucine..
Aleksandr
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The thing is though, most young guys have good hair but start losing it / thinning in their mid-late 20's and 30's, which age corresponds to a lot of professional sports people.
Where did you find those about methionine?