I wanted to get a thread started describing some of what I've been doing for the last 3 weeks... too soon to tell but the last 3 days or so have shown improvements so I wanted to share... and this is a thread in progress so I'll be adding links proving certain points but everyone interested can start doing their own research to see how this may fit in for them before trying anything like this... not that this is too far fetched but here's the start.
As we all know, Fin held DHT down which left estrogen unopposed ... high levels of estrogen then signal the pituitary gland to lower and even stop testosterone production (through LH, etc... ) ... as male genitalia decreases the testosterone production , the final result is hypothyroid. Primary hypothyroidism can reduce free testosterone levels and the opposite can be true.
What are symptoms of this? Weight gain the waist area, erectile dysfunction, reduced immunity to infections, brain fog... the list goes on. Much of this we know...
Another symptom though is high levels of Reverse T3... which is the anti-T3 and is produced from T4 (For the record, T4 is produced in the Thyroid and converted to either T3 or Reverse T3). This continues the downward spiral of hypo thyroid as a result of a stress state, infection or medications. (I have no proof FIN increases it directly but let's continue)
Hypothyroidism often causes elevated prolactin, which can lower testosterone levels. Elevated prolactin levels are caused by increased TRH, which is a response to underactive thyroid. This continues the assault on our manhood.
Also, Testosterone, DHT and 3adiolG fit the same cell receptor as Estradiol, but it is the first three that actually activate the cell, where as Estradiol binds but does not activate the cell receptor, stopping the first three from binding with the cell receptor... which is why we're not sensitive to certain hormones like DHT any longer...
During this stress state that we're in, the body chooses certain organs to limit blood flow to... sexual organs are last believe it or not to receive blood... which is why we suffer cold penis and ED. Our bodies are in a survival state and , for some, eventually clears but for others, the number of parts out of whack become overwhelming and don't recover naturally...
I'm currently researching why the 2nd timers on FIN have a higher propensity to PFS ... RT3 supposedly has a short half life and tests don't always prove high levels, but I'm not convinced something isn't sticking around far longer than a normal blood test can show. This is theory though... but some inflammation in our gut, for example, limited T3 conversion... we stayed in this state not knowing we had a low level issue & tried FIN again only to suffer more T3 losses and kick off the downward spiral.
So here's what I'm doing... since Reverse T3 is produced only by T4 and by taking T3, my own production of T4 drops limiting what can be converted, I'm taking 30mcg T3 Morning and Afternoon... I have a better body temperature & genitals appear fuller (measured fuller too). The tightness hasn't returned for 72 hours thus far. I have regular bowel movements as well. I'm planning 120 days at least and increasing T3 until I hit about 100Mcg and then cycling down.
I'm also taking approx .5mg Letro every 3 days. This is to lower estrogen and boost my own testosterone production. I've used Letro in this fashion many times before but I think the combo of this and returning blood flow is why i'm feeling better in the last 3 days... with Sunday and today showing signs of libido and a 'natural' erection... not quite random but certainly not my doing. Lower estrogen can kill libido so it's funny I'm seeing some of it but I'm not expecting much until Letro is completely removed.
Thyroid issues (no I'm not RPF, lol) track back to inflammation , fungal infections (I never had a toe fungal infection until Fin. I've asked others , some yes, some no so far) , brain fog, ED... and I think you have to address more of the endocrine system than just estrogen... some of this traces back to Gbold's original theory of lowering prolactin which is a biggie... and Androsterone use is also thyroid mimetic so that could be more of a reason why it's working than just the needed DHT.
I'll post more on here and always ready to share... but the symptoms of PFS and it's hormonal imbalance fit hand & hand with hypothryroid, reverse T3, high estrogen, etc... As I found more RT3 symptoms and how detrimental hypo is to all enzyme reactions, no wonder taking certain products did sh1t for us... or made us worse...
As we all know, Fin held DHT down which left estrogen unopposed ... high levels of estrogen then signal the pituitary gland to lower and even stop testosterone production (through LH, etc... ) ... as male genitalia decreases the testosterone production , the final result is hypothyroid. Primary hypothyroidism can reduce free testosterone levels and the opposite can be true.
What are symptoms of this? Weight gain the waist area, erectile dysfunction, reduced immunity to infections, brain fog... the list goes on. Much of this we know...
Another symptom though is high levels of Reverse T3... which is the anti-T3 and is produced from T4 (For the record, T4 is produced in the Thyroid and converted to either T3 or Reverse T3). This continues the downward spiral of hypo thyroid as a result of a stress state, infection or medications. (I have no proof FIN increases it directly but let's continue)
Hypothyroidism often causes elevated prolactin, which can lower testosterone levels. Elevated prolactin levels are caused by increased TRH, which is a response to underactive thyroid. This continues the assault on our manhood.
Also, Testosterone, DHT and 3adiolG fit the same cell receptor as Estradiol, but it is the first three that actually activate the cell, where as Estradiol binds but does not activate the cell receptor, stopping the first three from binding with the cell receptor... which is why we're not sensitive to certain hormones like DHT any longer...
During this stress state that we're in, the body chooses certain organs to limit blood flow to... sexual organs are last believe it or not to receive blood... which is why we suffer cold penis and ED. Our bodies are in a survival state and , for some, eventually clears but for others, the number of parts out of whack become overwhelming and don't recover naturally...
I'm currently researching why the 2nd timers on FIN have a higher propensity to PFS ... RT3 supposedly has a short half life and tests don't always prove high levels, but I'm not convinced something isn't sticking around far longer than a normal blood test can show. This is theory though... but some inflammation in our gut, for example, limited T3 conversion... we stayed in this state not knowing we had a low level issue & tried FIN again only to suffer more T3 losses and kick off the downward spiral.
So here's what I'm doing... since Reverse T3 is produced only by T4 and by taking T3, my own production of T4 drops limiting what can be converted, I'm taking 30mcg T3 Morning and Afternoon... I have a better body temperature & genitals appear fuller (measured fuller too). The tightness hasn't returned for 72 hours thus far. I have regular bowel movements as well. I'm planning 120 days at least and increasing T3 until I hit about 100Mcg and then cycling down.
I'm also taking approx .5mg Letro every 3 days. This is to lower estrogen and boost my own testosterone production. I've used Letro in this fashion many times before but I think the combo of this and returning blood flow is why i'm feeling better in the last 3 days... with Sunday and today showing signs of libido and a 'natural' erection... not quite random but certainly not my doing. Lower estrogen can kill libido so it's funny I'm seeing some of it but I'm not expecting much until Letro is completely removed.
Thyroid issues (no I'm not RPF, lol) track back to inflammation , fungal infections (I never had a toe fungal infection until Fin. I've asked others , some yes, some no so far) , brain fog, ED... and I think you have to address more of the endocrine system than just estrogen... some of this traces back to Gbold's original theory of lowering prolactin which is a biggie... and Androsterone use is also thyroid mimetic so that could be more of a reason why it's working than just the needed DHT.
I'll post more on here and always ready to share... but the symptoms of PFS and it's hormonal imbalance fit hand & hand with hypothryroid, reverse T3, high estrogen, etc... As I found more RT3 symptoms and how detrimental hypo is to all enzyme reactions, no wonder taking certain products did sh1t for us... or made us worse...
