Fighting….
- Thread starter bruschi11
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Sugar and iron make cortisol ultimately…..
I’m high in sugar (blood glucose) and low in iron. I guess it’s time for keto and possibly some short dry fasts.
I’m so weak. I don’t know how I’m gonna pull it off. It’s possible I try to stay on a little hormonal therapy test dhea as I keep moly up and start this approach.
Starting keto immediately after sending my OATs and hair in this week.
I’m high in sugar (blood glucose) and low in iron. I guess it’s time for keto and possibly some short dry fasts.
I’m so weak. I don’t know how I’m gonna pull it off. It’s possible I try to stay on a little hormonal therapy test dhea as I keep moly up and start this approach.
Starting keto immediately after sending my OATs and hair in this week.
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Big finding re: moly. Moly sulfating dhea during copper deficiency ends up lowering dhea. Cuz copper converts pregnenolone to dhea. So without copper… we give moly we kill dhea more which is already low in copper deficiency. And that locks iron and copper in liver. Cuz it’s dhea that makes iron and copper available from liver.
I was completely impotent the last two weeks while on test/moly Bs (without b12/b1/v3 as keeping nadph down is priority.)
I gave dhea and libido came back over night. Just about 20mg over two days.
I am so messed up still. I guess all I can really focus on is sulfite, while raising iron/sulfur.
While bringing in keto.
Which I will start by the weekend waiting to do some testing first.
I was completely impotent the last two weeks while on test/moly Bs (without b12/b1/v3 as keeping nadph down is priority.)
I gave dhea and libido came back over night. Just about 20mg over two days.
I am so messed up still. I guess all I can really focus on is sulfite, while raising iron/sulfur.
While bringing in keto.
Which I will start by the weekend waiting to do some testing first.
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Bringing in androsterone to accompany dhea. And continuing with test suspension 3mg E3d. So every 72 hours I guess.
Really digging back into hormones. And I just think it has to happen. Iron is #1 right now with moly. Need to down regulate nadph first and foremost.
I read more gbold today than I have in a long time. Hormone stuff. Nadph stuff. Hormones are a required approach here. But you gotta see g6pd runs on glucose. And if we can block glucose for now, nadph has to fall.
I also have one pill of RU486 on hand from 7 years ago lol. Considering using it. And if it goes decently then ordering some more. I might be too anemic for it currently. We are going to see as the long half life scares me .
Really digging back into hormones. And I just think it has to happen. Iron is #1 right now with moly. Need to down regulate nadph first and foremost.
I read more gbold today than I have in a long time. Hormone stuff. Nadph stuff. Hormones are a required approach here. But you gotta see g6pd runs on glucose. And if we can block glucose for now, nadph has to fall.
I also have one pill of RU486 on hand from 7 years ago lol. Considering using it. And if it goes decently then ordering some more. I might be too anemic for it currently. We are going to see as the long half life scares me .
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Completely shifting in another direction. I got pushed into hif1a the other day. I went into it hard. And as I realized with my nutritionist this is what is affecting me currently. Essentially overactive nrf2…. I realized- this is exactly what I treated in 2017 when I recovered. Obviously it was unknowingly but really everything I did was anti hif1a.
Hif1a raises cystine which went high in bloodwork 18 months ago. It pressures nadph. And I’ve been saying this “my high b12 in blood is being used (it activates hif1) and using up my b1 (nadph).
So hif1 overactivity forces nadph upregulation wasting b1 pressuring nadph oxidase (b2.)
More than that though it uses up copper more than anything, iron, zinc, manga, moly. It’s a disaster.
It raises carbon monoxide. It shrinks the brain hard.
Cholesterol (high) causes overactive hif1 next to the high b12. This is all clear.
Shifting to strict Okinawa diet and considering a juice fast.
Hif1a raises cystine which went high in bloodwork 18 months ago. It pressures nadph. And I’ve been saying this “my high b12 in blood is being used (it activates hif1) and using up my b1 (nadph).
So hif1 overactivity forces nadph upregulation wasting b1 pressuring nadph oxidase (b2.)
More than that though it uses up copper more than anything, iron, zinc, manga, moly. It’s a disaster.
It raises carbon monoxide. It shrinks the brain hard.
Cholesterol (high) causes overactive hif1 next to the high b12. This is all clear.
Shifting to strict Okinawa diet and considering a juice fast.
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RebelWithACause
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Never liked sweet potatoes for some reason. They also eat/ate a lot of pig.
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I’m gonna post my posts from RPF here too. I do believe I’m battling the Guam disease ALS- Parksinsons dementia complex. It’s very bad and due to its progression it’s far more about fixing the entire system than just the toxin bmaa.
It’s clear I’m just inflating superoxide like crazy as thyroid turns on/ selenium is used.
I used enclomiphene last night after testosterone suspension has been horrible the last few weeks. I had been using the test twice a week mainly only 2-3mg each time.
I can’t explain just how bad the other day was from test suspension. And then last night into this morning enclompiphene legit put me into the craziest estrogen/ copper deficiency imaginable.
Everywhere I was gettting to is that beta alanine needs copper and nrf2 system working together and zinc.
Beta alanine seems to be the key to co2 for me. But nrf2 system needs to go with it (V/ cr, ALa, E, c , coq10). And then there’s thyroid which these allow to turn on .
So there’s really 4 big pieces here.
SOD: cu, zn, mn in that order is #1. This is what we are trying to fix end of day. I think this truly is ALS at its core and this is the end goal.
Co2 : beta alanine / b5 , nrf2 system to help get pyruvate dehydrogenase up and working. Using my pyruvate (provided by SOD) to make co2 in the Krebs.
Thyroid: beta alanine for tyrosine then iodine to get t4 up and made. To use selenium. And get sulfur in charge so I can sulfate dhea. Unfortunately, thyroid is destroying estrogen. So the goal is to really use up the high selenium so t3 isn’t so harsh on me.
FAD/nadph/ active b6/ nad system: affects all of the above. Co2, electrolyte retention, sod, thyroid. I mean it’s part of everything it just comes first.
Need to keep vitamin A down but can’t do another liver flush for another 5 days or so. This low is just lower than ever.
I just can’t believe a guy this sick was handled the way I was. Mental institutions etc.
This is hell.
It’s clear I’m just inflating superoxide like crazy as thyroid turns on/ selenium is used.
I used enclomiphene last night after testosterone suspension has been horrible the last few weeks. I had been using the test twice a week mainly only 2-3mg each time.
I can’t explain just how bad the other day was from test suspension. And then last night into this morning enclompiphene legit put me into the craziest estrogen/ copper deficiency imaginable.
Everywhere I was gettting to is that beta alanine needs copper and nrf2 system working together and zinc.
Beta alanine seems to be the key to co2 for me. But nrf2 system needs to go with it (V/ cr, ALa, E, c , coq10). And then there’s thyroid which these allow to turn on .
So there’s really 4 big pieces here.
SOD: cu, zn, mn in that order is #1. This is what we are trying to fix end of day. I think this truly is ALS at its core and this is the end goal.
Co2 : beta alanine / b5 , nrf2 system to help get pyruvate dehydrogenase up and working. Using my pyruvate (provided by SOD) to make co2 in the Krebs.
Thyroid: beta alanine for tyrosine then iodine to get t4 up and made. To use selenium. And get sulfur in charge so I can sulfate dhea. Unfortunately, thyroid is destroying estrogen. So the goal is to really use up the high selenium so t3 isn’t so harsh on me.
FAD/nadph/ active b6/ nad system: affects all of the above. Co2, electrolyte retention, sod, thyroid. I mean it’s part of everything it just comes first.
Need to keep vitamin A down but can’t do another liver flush for another 5 days or so. This low is just lower than ever.
I just can’t believe a guy this sick was handled the way I was. Mental institutions etc.
This is hell.
RebelWithACause
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It sucks when you are sick the world keeps spinning. Society doesn't give a shit about sick people. Sickness/weakness = cast away because weakness means a liability. For most people. Maybe not everyone.
Although there are people out there who care (to the extent it is possible/realistic) But often their empathy was created through their own struggle with sickness. I have much more empathy for sick people and older people than before I had this stuff. Makes you realise how many people get the short end of the stick.
Same for how you were handled and put in mental institution. It's a way to cast away and hide the problem. Most people don't understand anything about mental illness. Or body chemistry. So in that sense it is logical they put you on drugs. They don't know better
Although there are people out there who care (to the extent it is possible/realistic) But often their empathy was created through their own struggle with sickness. I have much more empathy for sick people and older people than before I had this stuff. Makes you realise how many people get the short end of the stick.
Same for how you were handled and put in mental institution. It's a way to cast away and hide the problem. Most people don't understand anything about mental illness. Or body chemistry. So in that sense it is logical they put you on drugs. They don't know better
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Co2 absorbs boron which turns d receptor on allowing calcium absorption and metabolism to work.
Controlling nitric oxide allows body to absorb iodine.
Then we need to make tyrosine for t4 next to iodine.
This all brings selenium down and brings sulfur up so we can sulfate dhea.
Really need fad / active b6 for this. So we can get chloride into the cells and retain our potassium.
Controlling nitric oxide allows body to absorb iodine.
Then we need to make tyrosine for t4 next to iodine.
This all brings selenium down and brings sulfur up so we can sulfate dhea.
Really need fad / active b6 for this. So we can get chloride into the cells and retain our potassium.
RebelWithACause
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A bit off topic but the Ray Peat Forum (Low Toxin Forum) seems fairly dead now. Eventhough Ray Peat was wrong with certain things that forum was a good reflection of how Ray Peat thought and I found a lot of information and people there that helped me get better. Shame to see it in such state. It was a good time though. Thankful for it being there. It was like a crossroads of different people. Pretty sure Ray Peat didn't even endorse that forum.
I think his death also made people kind of walk away from it.
I think his death also made people kind of walk away from it.
Yeah I am not really interested much about whats posted there atm, but they still discuss other topics even with the focus being low vit A, the people who didn't like the change created this forum:
Bioenergetic Forum
bioenergetic.forum
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I think it’s dht that plays a big role in making t4 for men. And 7-keto-dhea for making t3.
Need tyrosine for t4. Does dht keep bh4 cycle up for tyrosine? I think so. Or it just maintains the enzyme tyrosine hydroxylase which breaks tyrosine down.
7-keto - dhea seems to me uses selenium for t3.
Need tyrosine for t4. Does dht keep bh4 cycle up for tyrosine? I think so. Or it just maintains the enzyme tyrosine hydroxylase which breaks tyrosine down.
7-keto - dhea seems to me uses selenium for t3.
RebelWithACause
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It's interesting how these hormones help with conversions and retentions of minerals.
Might explain why when I used finasteride I felt lower cortisol and lower thyroid in general. It slows down my metabolism. Lowers adrenaline as well. I don't feel good on it because of this. DHT is amazing if it's in good range.
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Copper deficiency (finasteride) lowers dhea and 7 keto dhea. No dhea no copper iron available from liver. No 7 keto dhea, body can’t use selenium so no thyroid.
Death drug. Silly. Sad. Lives destroyed.
8 year anniversary for me taking it in a few weeks.
One last push to save this life. I’m moving to a beautiful place by the ocean in two days. I got a great life ahead. I will be a nutritional consultant type for career one day I guess. Might be hard to make money and I don’t want to take money from the sick.
That stuff whatever. What matters is saving this brain saving this life.
RebelWithACause
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It lowers all DHT metabolites as well! Like Gbolduev said.
I know a few people (online) on it. Some of them do alright (read: alright, not great). They seemed to be decently healthy before using this drug. Definitely had some issues causing mild hairloss/thinning. They retain a masculine look. These guys seem to handle the drug ok.
Then have others who had health issues before finasteride. Fairly big health issues. They seem to go downhill fast and get the feminization effect the most. They are also often young looking before using it with a weaker puberty prior to using.
That being said ALL of them have something affected negatively by it. Differentiated between percentages. E.g. one of them who does alright on it definitely said he had lower sex drive and also lower drive and enjoyment of life.
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T4 and fad go toghether. No doubt in my mind. Making t4 or even taking t4 raises fad. T3 we know raises fmn.
Have to realize that FAD is key to electrolyte retention in the cell.
B12 takes down fad. Selenium takes down t4.
T3 takes down progesterone.
Look how everything affects everything here
Need fad for b12 usage to raise nadph.
Need fad for chloride in the cell. BUT high b12 kills chloride. Uses all your chloride to raise hif1a. Hif1a also takes down iron and copper and zinc. Hif1a killls Ceruloplasmin and A metabolism. Hence the iron copper issues
THAT is where high b12 fucks everyone up in cfs. And why cfs is so intricate.
Soooo many people have high b12 in cfs. Then soooo many people have low iodine in oligoscans . Then a lot have high selenium in oligoscans.
We need to get b12 and selenium normalized. These very likely are the two deciding factors in my case.
I don’t know if it’s too late. Years have passed. Years. My mitochondria is so fucked up. But my recoveries reflect this.
Won’t go any deeper. Need to get in control here then explain. If I already lost. And I can’t get better. What a shame that would be.
Have to realize that FAD is key to electrolyte retention in the cell.
B12 takes down fad. Selenium takes down t4.
T3 takes down progesterone.
Look how everything affects everything here
Need fad for b12 usage to raise nadph.
Need fad for chloride in the cell. BUT high b12 kills chloride. Uses all your chloride to raise hif1a. Hif1a also takes down iron and copper and zinc. Hif1a killls Ceruloplasmin and A metabolism. Hence the iron copper issues
THAT is where high b12 fucks everyone up in cfs. And why cfs is so intricate.
Soooo many people have high b12 in cfs. Then soooo many people have low iodine in oligoscans . Then a lot have high selenium in oligoscans.
We need to get b12 and selenium normalized. These very likely are the two deciding factors in my case.
I don’t know if it’s too late. Years have passed. Years. My mitochondria is so fucked up. But my recoveries reflect this.
Won’t go any deeper. Need to get in control here then explain. If I already lost. And I can’t get better. What a shame that would be.
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To add to the above^^^
ALA allows iodine absorption. I’ve been saying t4 =fad. Well there you have it. ALA allowing iodine absorption leads to FAD….
Cutler cured autism for a reason. He was fixing FAD.
Cutler “people only fix copper if they take ALA with my approach.” FAD to me is how you end up fixing copper.
ALA allows iodine absorption. I’ve been saying t4 =fad. Well there you have it. ALA allowing iodine absorption leads to FAD….
Cutler cured autism for a reason. He was fixing FAD.
Cutler “people only fix copper if they take ALA with my approach.” FAD to me is how you end up fixing copper.